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Gamma-aminobutyric-acid (GABA) agonists for neuroleptic-induced tardive dyskinesia (TD)

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Gamma-aminobutyric-acid (GABA) agonists for neuroleptic-induced tardive dyskinesia (TD)

Sübutlu məlumatların xülasələri
04.09.2017 • Sonuncu dəyişiklik 04.09.2017
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GABA agonists (baclofen, progabide, sodium valproate, tetrahydroisoxazolopyridine) may cause several adverse effects, and their effectiveness in people with antipsychotic-induced tardive dyskinesia is inconclusive and unconvincing.

Eight poorly reported studies were included in an update of a Cochrane review . Patients with schizophrenia or other chronic mental illnesses with signs of antipsychotic-induced TD were included. For the outcome of ’no clinically important improvement in tardive dyskinesia’ GABA agonist drugs were not clearly better than placebo (n = 108, RR 0.83 CI 0.6 to 1.1). Deterioration in mental state was more likely to occur in people receiving GABA medication (n = 95, RR 2.47 CI 1.1 to 5.4). There is a suggestion of an increase in ataxia (loss of power of muscular coordination) for both baclofen and sodium valproate (n = 95, RR 3.26 CI 0.4 to 30.2), and in sedation (n = 113, RR 2.12 CI 0.8 to 5.4) compared with placebo, but this was not significant.

Comment: The quality of evidence is downgraded by study quality (inadequate allocation concealment), imprecise results (limited study size for each comparison).

Ədəbiyyat

  1. Alabed S, Latifeh Y, Mohammad HA et al. Gamma-aminobutyric acid agonists for neuroleptic-induced tardive dyskinesia. Cochrane Database Syst Rev 2011;(4):CD000203.